What statement about the mechanism of hunger and satiety is true? Neuropeptide Y plays a powerful role in signaling satiety.

The small intestines release the hormone cholecystokinin, which encourages us to eat.

Fat cells release leptin, which travels to the brain to signal the need to eat.

The ventromedial hypothalamus plays a role in satiety.​

The correct answer is: The ventromedial hypothalamus plays a role in satiety.​

Explanation:

The hypothalamus is a part of the brain that controls many important bodily functions and connects both the nervous system with the endocrine system. The hypothalamus consists of several nuclei that have diverse functions and are located in 3 different regions.

The nucleus that plays a role in satiety is the ventromedial nucleus, terminating hunger and giving a sensation of fullness. It also plays a significant role in thermoregulation, among other things.

Neuropeptide Y, on the other hand, is a peptide that is released to make us feel hungry and encourage us to intake food (primarily carbohydrates).

Cholecystokinin is a hormone released by the small intestines after we had a meal, and its function is to improve digestion and make us feel full.

Fat cells DO release leptin, but the function of this hormone is to produce satiety, by stimulating anorexigenic (meaning they take hunger away) hormones and inhibiting orexigenic ones, like Neuropeptide Y.

Mitochondria

Explanation:

The mitochondria creates energy for the cell by converting glucose to ATP energy.